Magnesium is one of those nutrients that can sound simple on the surface. It gets mentioned for cramps, sleep, stress and muscle relaxation, so it is easy to think of it as a basic supplement you take when you feel tight, tired or wired.

But magnesium is much deeper than that, and in this article, we’ll unpack magnesium from various angles.

Magnesium is an essential mineral involved in more than 300 enzymatic reactions in the body. It supports energy production, protein synthesis, nerve communication, muscle contraction and relaxation, blood glucose regulation, blood pressure regulation, DNA function, electrolyte balance and normal cellular signalling (Kirkland et al., 2018; DiNicolantonio et al., 2018).

That is why magnesium is not just a sleep nutrient, a cramp nutrient or a recovery nutrient. It is a foundational mineral for normal physiology.

Magnesium becomes especially relevant when the body has been under pressure. Poor diet, heavy alcohol use, chronic stress, hard training, inconsistent meals, digestive issues and poor sleep can all increase the need to pay attention to magnesium intake and status.

Whether someone is rebuilding after alcohol use, dealing with long-term stress, training hard, eating inconsistently, struggling with sleep or simply trying to understand their nutrition better, magnesium is worth understanding properly.

What To Know (Snapshot Takeaway)

• Magnesium is an essential mineral and cofactor in hundreds of enzymatic reactions.
• It plays a major role in ATP production, nerve signalling, muscle relaxation, blood glucose regulation, blood pressure regulation, DNA stability and neurotransmission.
• Magnesium is especially relevant to the nervous system because it helps regulate excitatory signalling, including pathways involving glutamate and NMDA receptors (Kirkland et al., 2018; Gillessen et al., 2000-2013).
• Low magnesium intake is not rare. Older Australian Health Survey data found inadequate magnesium intake was common in younger Australians (Australian Bureau of Statistics, 2015). More recent ABS data also suggests inadequate magnesium intake remains common across the population (Australian Bureau of Statistics, 2026).
• Alcohol, poor dietary intake, digestive issues, diuretics, high stress, diabetes, diarrhoea and some medications can all increase the likelihood that magnesium status needs attention.
• Food should come first where possible, but supplement form matters. Magnesium glycinate, citrate, oxide, malate, taurate, threonate, chloride and orotate are not the same.
• Do not treat symptoms alone as proof of deficiency. Magnesium assessment should consider diet, symptoms, medications, health conditions and testing where appropriate.

What Is Magnesium?

Magnesium, or Mg, is an essential mineral. Your body cannot make it, so it has to come from food, fluids and, in some cases, supplements.

Most magnesium is stored in bone and muscle. Only a small amount circulates in the blood, which is one reason magnesium deficiency can be difficult to assess from a standard blood test alone. This does not mean testing is pointless. It means the result needs to be interpreted with the broader clinical picture.

Magnesium is involved in protein synthesis, cellular energy production, glucose metabolism, blood pressure regulation, neuromuscular function and nervous-system signalling (DiNicolantonio et al., 2018; Kirkland et al., 2018).

One of the deeper reasons magnesium matters is its relationship with the nervous system.

Magnesium helps regulate excitatory signalling in the brain and nervous system, particularly through its influence on NMDA receptors and glutamate pathways (Kirkland et al., 2018). Glutamate is an important excitatory neurotransmitter. We need it for normal brain function, but excessive excitatory activity is not something we want running unchecked.

In more technical terms, high excitatory amino acid activity can contribute to neurotoxicity when the system is pushed beyond its normal range (Gillessen et al., 2000-2013). In more practical language: magnesium is one of the nutrients involved in keeping nerve signaling from becoming too overexcited.

That does not mean magnesium is a treatment for anxiety, addiction, insomnia or neurological disease. It means magnesium sits right in the middle of many systems that matter when someone feels stressed, depleted, wired, tense, crampy, sleep-deprived or under-recovered.

The Role of Magnesium in the Body

Magnesium serves many purposes in the body. The list can get long very quickly, but the main roles are worth understanding.

One key role is ATP production. ATP, or adenosine triphosphate, is the main energy currency your cells use to do work. Magnesium is required for normal ATP function, so when we talk about cellular energy, magnesium is part of that conversation (DiNicolantonio et al., 2018).

Magnesium also supports:

• Protein synthesis: Magnesium acts as a cofactor for enzymes involved in RNA, DNA and protein synthesis.
• Muscle contraction and relaxation: Magnesium interacts with calcium signalling, which helps muscles contract and relax appropriately.
• Neurons and nerve function: Magnesium is involved in neurotransmitter release and normal nerve communication (Kirkland et al., 2018).
• Blood glucose control: Magnesium plays a role in insulin action and glucose metabolism (DiNicolantonio et al., 2018).
• Blood pressure regulation: Magnesium can influence vascular tone and calcium-channel activity, which is relevant to blood pressure physiology.
• Hormone receptor signalling: Magnesium helps support normal cellular signalling processes.
• Calcium-channel gating: Magnesium influences calcium movement across membranes, which matters for nerve and muscle function.
• DNA support: Magnesium contributes to DNA stability, repair and replication.
• Glycolysis: Magnesium is involved in enzymes that help break down glucose for energy.

So when someone says magnesium is important, they are not exaggerating. It is woven through normal body function.

The practical takeaway is this: if your diet has been poor, your stress has been high, your sleep has been rough, you have been drinking heavily, your digestion is compromised or you are relying on ultra-processed foods, magnesium is one of the first nutrients worth bringing back into the conversation.

Magnesium, Alcohol and Nutrient Depletion

Although this article is not focused on alcohol and addiction, it’s part of my past experience, and deserves it’s own section due to many ways alcohol can impact magnesium status.

Alcohol can contribute to magnesium depletion through several direct and indirect mechanisms.

Reduced absorption

Chronic alcohol intake can affect the gastrointestinal tract and may impair normal nutrient absorption. Magnesium is absorbed mainly in the small intestine, so gut integrity and digestive function matter.

Increased loss

Alcohol can have a diuretic effect, increasing urine production and potentially increasing the loss of minerals and electrolytes. Magnesium can be lost this way, particularly when alcohol intake is frequent or heavy.

Poor dietary habits

Alcohol often displaces food. Some people skip meals, under-eat, rely on takeaway, crave refined carbohydrates or lose the routine of balanced eating. That can mean fewer magnesium-rich foods such as nuts, seeds, legumes, leafy greens, whole grains and quality protein meals.

Wider metabolic stress

Alcohol can also affect liver function, hormones, sleep, blood sugar and appetite regulation. These do not all mean magnesium deficiency is guaranteed, but they do increase the relevance of nutrition assessment.

This is why magnesium can matter in alcohol reduction or recovery. Not because magnesium fixes recovery, but because the body may need better nutritional foundations after a long period of depletion.

How Do I Know If I Am Low in Magnesium?

It’s a good question, one that is asked frequently. Magnesium deficiency can be hard to identify from symptoms alone.

As mentioned earlier, most magnesium is stored in bone and muscle, with only a small amount in the blood. That can make standard blood testing an incomplete picture. Urine testing, red blood cell magnesium, hair mineral analysis or broader metabolic testing may be considered in some clinical contexts, but no single test should be treated as perfect.

The better approach is to look at the whole picture:

• What does your diet look like?
• Are you eating magnesium-rich foods regularly?
• Do you drink alcohol frequently or heavily?
• Are you under high stress?
• Do you have digestive symptoms?
• Are you taking medications that may affect magnesium?
• Are there signs of electrolyte disturbance?
• Are symptoms ongoing, severe or unexplained?

Older Australian Health Survey data found inadequate magnesium intake was common, especially among younger Australians (Australian Bureau of Statistics, 2015). More recent data from the ABS also shows inadequate magnesium intake remains a meaningful issue in Australia (Australian Bureau of Statistics, 2026).

That does not mean everyone needs a supplement. It does mean magnesium intake deserves more attention than it usually gets.

Common Factors That Can Increase Magnesium Risk

Several factors can increase the chance that magnesium intake, absorption or retention may be compromised.

These include:

• Excessive use of diuretics (Caffeine, stimulants, etc): Increases urinary excretion, leading to loss of magnesium.
• Alcohol: Impairs absorption and increases excretion of magnesium.
• Hypochlorhydria (low stomach acid): Reduces magnesium absorption efficiency.
• Antacids: Bind to and reduce absorption of magnesium.
• Vitamin D (due to its role in Calcium absorption): Alters calcium and magnesium balance, potentially decreasing magnesium levels.
• Coeliac disease: Damages the intestinal lining, hindering magnesium absorption.
• Chronic stress (emotional or psychological – overactive sympathetic nervous system): Elevates stress hormones, increasing magnesium excretion.
• Crohn’s disease: Causes malabsorption of nutrients, including magnesium.
• Type 1 and 2 Diabetes: Increases urinary loss of magnesium.
• Diarrhoea / Laxatives: Leads to rapid loss of fluids and electrolytes, including magnesium.
• Diets high in sugar: Require more magnesium for processing, depleting body stores.
• Excessive menstruation: Can lead to increased loss of magnesium.
• High phosphorus in the diet (inorganic phosphates found in inactive processed food ingredients): Competes with and reduces magnesium absorption.
• Insulin resistance: Linked to lower magnesium levels in the body.
• Low salt intake: May affect magnesium levels due to the interconnected balance of electrolytes.
• Low selenium intake: Selenium deficiency can affect magnesium status and metabolism.
• Vitamin B6 (pyridoxine) deficiency: Essential for magnesium absorption and utilisation in the body.

Don’t use this list as gospel, but if you notice you tick off some of these, magnesium intake might be something to consider.

If someone has poor magnesium intake, multiple risk factors plus symptoms, magnesium is worth assessing properly.

Potential Signs of Magnesium Deficiency

Signs of magnesium deficiency can vary. Some can be vague. Some can be serious. Many overlap with other conditions.

One clinical clue to consider is unexplained hypokalaemia, or low potassium, and hypocalcaemia, or low calcium. These can sometimes point toward significant magnesium depletion. Neuromuscular irritability, including signs such as the Trousseau sign, may also be relevant in more significant depletion states (Pokan et al., 2006).

Possible signs associated with low magnesium or altered magnesium status can include:

• Muscle cramps
• Muscle weakness
• Twitching or fasciculations
• Tremors
• Irritability
• Anxiety-like symptoms
• Poor sleep
• Fatigue
• Neuromuscular irritability
• Tinnitus
• Disorientation in more significant cases
• Abnormal heart rhythm in more severe deficiency states
• Low calcium or low potassium on testing

More severe deficiency states can be associated with more serious features such as arrhythmias, convulsions or marked neuromuscular symptoms. These require medical assessment, not supplement guessing.

Important to remember that symptoms are not always proof, and investigations are always recommended to confirm as well.

Choosing the Correct Magnesium Supplement

Not all magnesium supplements are created equal.

If a product has magnesium written on the front label, that does not tell you the full story. The form of magnesium matters. The dose of elemental magnesium matters. The person’s digestion, goal, medication use and tolerance all matter.

On a slightly more technical level, magnesium is bound to another compound in supplement form. That companion compound affects absorption, tolerance and how the supplement behaves in the gut.

This is why magnesium citrate is different from magnesium oxide, magnesium glycinate is different from magnesium malate, and magnesium threonate is different again.

Magnesium glycinate

Magnesium glycinate is magnesium bound to glycine. It is often chosen when sleep, nervous-system support or bowel tolerance are priorities.

Research comparing magnesium diglycinate with magnesium oxide in people with ileal resection found that magnesium diglycinate showed better bioavailability in that specific clinical setting (Schuette et al., 1994).

Glycine itself has also been studied for sleep quality. This does not prove magnesium glycinate is a sleep cure, but it helps explain why this form is commonly discussed in sleep and calming contexts (Yamadera et al., 2007).

Magnesium oxide

Magnesium oxide is common, cheap and high in elemental magnesium, but it is generally less bioavailable than some other forms. Because more of it may stay in the gut, it can have a stronger laxative effect for some people (Schuchardt & Hahn, 2017).

That does not make it useless. It means it is often better suited to bowel-related use than to correcting low magnesium status quickly.

Magnesium citrate

Magnesium citrate is commonly used and generally better absorbed than oxide. It may also loosen stools, so dose and tolerance matter.

One trial has looked at oral magnesium for pregnancy-induced leg cramps (Supakatisant & Phupong, 2012). This is useful context, but it should not be stretched into a blanket claim that magnesium citrate fixes all cramping.

Magnesium chloride

Magnesium chloride is another form used in supplements and topical products. It may be considered where digestion, absorption or tolerance are priorities, although the quality of the product and the dose still matter.

Magnesium malate

Magnesium malate is magnesium bound to malic acid. Malate is involved in energy metabolism, which is why this form is often marketed around energy and muscle pain.

Malate-related cellular energy research helps explain why malate is discussed in energy-metabolism contexts (Scheibe, 2004). That reference is not direct proof that magnesium malate treats fatigue, but it gives useful biochemical context.

Magnesium taurate

Magnesium taurate is magnesium bound to taurine. Taurine has been studied in relation to cardiovascular function and cellular regulation (Zulli, 2011).

This form is often positioned as calming or heart-supportive, but claims should be kept sensible. It may be a useful option for some people, but it is not automatically the best magnesium for everyone.

Magnesium threonate

Magnesium threonate is often marketed around brain health because of interest in brain magnesium levels, memory and cognition.

Animal research has shown that elevating brain magnesium could affect synaptic loss and cognitive deficits in an Alzheimer’s disease mouse model (Li et al., 2014). NMDA receptor research in neuropathic pain contexts also helps explain why magnesium and excitatory signalling are often discussed together (Wu & Zhuo, 2009).

This is interesting, but it needs careful wording. Mouse studies and mechanistic papers are not the same as proving a human supplement outcome. Magnesium threonate may be worth watching, but it should not be oversold.

Magnesium sulfate

Magnesium sulfate is better known as Epsom salts. It is commonly used externally in baths. I would not frame it as a detox product, because that language often runs ahead of the evidence. If used, it is better discussed around relaxation rituals, muscle comfort and bath use rather than internal supplementation.

Magnesium orotate

Magnesium orotate is often discussed in cardiovascular and performance contexts. Research on oral magnesium therapy in coronary artery disease patients has looked at exercise heart rate, exercise tolerance and myocardial function (Pokan et al., 2006).

This is not a reason to self-prescribe magnesium for heart disease. It is a reason to respect magnesium’s role in cardiovascular physiology and seek proper guidance when heart health is involved.

Magnesium carbonate

Magnesium carbonate is often used for its antacid properties. Research on an aluminium- and magnesium-containing antacid has shown that magnesium-containing products can affect drug bioavailability in some contexts (Krishna et al., 2007).

This is a reminder that magnesium-containing antacids and medications can interact with absorption and medication timing. If you use medications, especially antibiotics or other timing-sensitive medicines, check with your practitioner or pharmacist.

Forms I am more cautious with

I am cautious about making blanket statements that a form is bad for everyone, but there are some forms I would approach carefully.

Magnesium hydroxide is commonly used as a laxative or antacid form and is not usually the form I would reach for when the goal is broader magnesium repletion.

Magnesium aspartate and magnesium pidolate can raise questions because they are bound to excitatory amino acid-related compounds. Excitatory amino acid neurotoxicity research gives some context for that caution (Gillessen et al., 2000-2013). The cautious framing is this: if someone already feels wired, over-stimulated or neurologically sensitive, I would usually choose a better-tolerated form first rather than defaulting to these.

Food Sources of Magnesium

Food should always be part of the magnesium conversation. While we can’t always get it from diet, a food first is generally always recommended to support foundational nutrient status.

Supplementation can be useful when there is a clear reason, but a food-first approach usually gives you more than magnesium. You also get fibre, potassium, polyphenols, healthy fats, protein, B vitamins and other minerals.

Good magnesium-rich foods include:

• Pumpkin Seeds – 535mg
• Raw Cacao – 507mg (Cacao nibs anyone?)
• Flax Seeds – 392mg
• Brazil Nuts – 350mg (also super high in selenium)
• Sesame Seeds – 340mg
• Chia Seeds – 335mg
• Almonds – 260mg
• Cashews – 250mg
• Buckwheat – 221mg
• Peanuts – 160mg
• Walnuts – 150mg
• Dark Chocolate (70%+) – 120mg (The darker the higher the Magnesium content)
• Tofu – 74mg
• Spinach – 74mg
• Sardines– 39mg
• Kale – 33mg
• Avocado – 27mg
• Whole Oats – 24mg

These foods are often compared per 100 g, which is useful for nutrient tables, but it is also worth being practical. You are probably not eating 100 g of pumpkin seeds in one sitting. A better way to use this list is to build magnesium-rich foods into daily meals.

For example:

• Add chia seeds or flax seeds to oats.
• Use pumpkin seeds over salads or yoghurt bowls.
• Eat almonds, cashews or walnuts as a snack.
• Include tofu, legumes or buckwheat in meals.
• Use spinach or kale in eggs, smoothies, soups or stir-fries.
• Choose dark chocolate as a magnesium-containing treat rather than a daily main source.

One important note: some plant foods contain phytates, also called phytic acid. Phytates can bind minerals and reduce absorption. This does not make nuts, seeds, legumes or grains bad. It simply means soaking, sprouting, fermenting, cooking and eating a varied diet can help improve overall mineral availability.

Correct Dosing for Magnesium Deficiency

Magnesium dosing needs care.

It depends on the person, the form, the reason for using it, the dose of elemental magnesium, bowel tolerance, kidney function, medications, pregnancy status and whether there is a confirmed or suspected deficiency.

DiNicolantonio et al. discuss magnesium deficiency and higher weight-based intake considerations in the context of subclinical deficiency and cardiovascular risk (DiNicolantonio et al., 2018). That is useful context, but it should not be turned into a general public dosing instruction.

When supplementing Magnesium, the general guidance is to:

• Check the elemental magnesium amount on the label.
• The average daily supplementation of elemental magnesium is 400-600 mg daily.
• Do not assume a higher dose is better.
• Be careful if magnesium causes loose stools, nausea or cramping.
• Avoid high-dose supplementation without guidance if you have kidney disease, heart rhythm issues, significant medical conditions or multiple medications.
• If pregnancy is involved, dosing should be discussed with a qualified practitioner.
• If a true deficiency is suspected, assess properly rather than guessing.

The phrase elemental magnesium matters. It refers to the true amount of magnesium in the supplement, not the total weight of the compound.

For example, 1000 mg of magnesium glycinate does not mean 1000 mg of elemental magnesium. The label should tell you the elemental magnesium per serve.

How To Choose a Magnesium Product

There are many magnesium products on the market, and some are much better formulated than others.

Rather than listing products here, I would focus on what to look for:

• Clear elemental magnesium amount per serve
• A form that matches the goal and digestive tolerance
• Minimal unnecessary fillers
• A dose that does not cause bowel irritation
• Practitioner guidance if you are using it for a specific condition
• No exaggerated claims around detox, cure-all effects or guaranteed sleep outcomes

A good product should make the dose and form clear. If you cannot tell what form of magnesium is being used, or how much elemental magnesium you are getting, I would be cautious.

The Takeaway

Magnesium is a critical mineral for normal human function. It is involved in energy production, nerve communication, muscle relaxation, glucose metabolism, DNA support, blood pressure physiology, electrolyte balance and nervous-system regulation.

It becomes especially relevant when someone has been under long-term stress, eating poorly, drinking heavily, sleeping badly, training hard, using diuretics or dealing with digestive issues.

The big picture is simple: magnesium matters because it supports the systems that help you feel steady, energised, relaxed, coordinated and resilient.

Start with food. Build meals around magnesium-rich ingredients such as nuts, seeds, leafy greens, legumes, tofu, whole grains, oats, cacao and quality whole foods. If supplementation is needed, choose the form carefully, pay attention to elemental magnesium and avoid assuming more is better.

If you are unsure whether magnesium is relevant for you, Stephen can help you look at your diet, symptoms, alcohol intake, stress load, sleep, training, pathology and supplement options in context.

FAQ

What does magnesium do in the body?

Magnesium supports hundreds of enzymatic reactions, including energy production, protein synthesis, nerve function, muscle contraction and relaxation, glucose metabolism, blood pressure regulation and DNA stability.

Why is magnesium important for the nervous system?

Magnesium helps regulate nerve signaling and excitatory pathways, including mechanisms involving glutamate and NMDA receptors (Kirkland et al., 2018). This is one reason it is often discussed in relation to stress, sleep, tension and nervous-system regulation.

Can alcohol lower magnesium?

Alcohol can contribute to low magnesium status through poor dietary intake, impaired absorption, increased urinary loss and wider metabolic stress. This is especially relevant with frequent or heavy alcohol intake.

How do I know if I am magnesium-deficient?

You cannot confirm magnesium deficiency from symptoms alone. Diet, symptoms, alcohol intake, digestive health, medications, pathology and sometimes additional testing need to be considered together.

What are signs of low magnesium?

Possible signs include cramps, twitching, fatigue, muscle weakness, irritability, poor sleep, tremors, neuromuscular irritability and, in more serious cases, abnormal heart rhythm or electrolyte disturbances. These signs can have many causes, so they should be assessed properly.

What is the best form of magnesium?

There is no single best form for everyone. Glycinate is often used for tolerance and sleep-focused support, citrate is common but can loosen stools, oxide is cheaper but generally less bioavailable, malate is often discussed around energy, and threonate is marketed around brain health. The best option depends on the person.

What does elemental magnesium mean?

Elemental magnesium is the actual amount of magnesium in the supplement. The total compound weight is different from the elemental magnesium dose, so always check the label.

Should I take magnesium every day?

Some people benefit from daily magnesium supplementation, but it is not automatically needed. Food intake, symptoms, health conditions, medications and dose all matter. If you are unsure, get individual guidance.

References

Australian Bureau of Statistics. (2015). Australian Health Survey: Usual nutrient intakes, 2011-12 financial year. https://www.abs.gov.au/statistics/health/health-conditions-and-risks/australian-health-survey-usual-nutrient-intakes/latest-release

Australian Bureau of Statistics. (2026). Usual nutrient intakes, 2023. https://www.abs.gov.au/statistics/health/health-conditions-and-risks/usual-nutrient-intakes/latest-release

DiNicolantonio, J. J., O’Keefe, J. H., & Wilson, W. (2018). Subclinical magnesium deficiency: A principal driver of cardiovascular disease and a public health crisis. Open Heart, 5(1), e000668. https://doi.org/10.1136/openhrt-2017-000668

Gillessen, T., Budd, S. L., & Lipton, S. A. (2000-2013). Excitatory amino acid neurotoxicity. In Madame Curie Bioscience Database. Landes Bioscience. https://www.ncbi.nlm.nih.gov/books/NBK6108/

Kirkland, A. E., Sarlo, G. L., & Holton, K. F. (2018). The role of magnesium in neurological disorders. Nutrients, 10(6), 730. https://doi.org/10.3390/nu10060730

Krishna, G., Kisicki, J. C., Olsen, S., Grasela, D. M., & Wang, Z. (2007). Effect of an aluminum- and magnesium-containing antacid on the bioavailability of garenoxacin in healthy volunteers. Pharmacotherapy, 27(7), 963-969. https://doi.org/10.1592/phco.27.7.963

Li, W., Yu, J., Liu, Y., Huang, X., Abumaria, N., Zhu, Y., Huang, X., Xiong, W., Ren, C., Liu, X., Chui, D., & Liu, G. (2014). Elevation of brain magnesium prevents synaptic loss and reverses cognitive deficits in Alzheimer’s disease mouse model. Molecular Brain, 7, 65. https://doi.org/10.1186/s13041-014-0065-y

Pokan, R., Hofmann, P., von Duvillard, S. P., Smekal, G., Wonisch, M., Lettner, K., Schmid, P., Shechter, M., Silver, B., Bachl, N., & Schmid, P. (2006). Oral magnesium therapy, exercise heart rate, exercise tolerance, and myocardial function in coronary artery disease patients. British Journal of Sports Medicine, 40(9), 773-778. https://doi.org/10.1136/bjsm.2006.027250

Scheibe, R. (2004). Malate valves to balance cellular energy supply. Physiologia Plantarum, 120(1), 21-26. https://doi.org/10.1111/j.0031-9317.2004.0222.x

Schuchardt, J. P., & Hahn, A. (2017). Intestinal absorption and factors influencing bioavailability of magnesium: An update. Current Nutrition & Food Science, 13(4), 260-278. https://doi.org/10.2174/1573401313666170427162740

Schuette, S. A., Lashner, B. A., & Janghorbani, M. (1994). Bioavailability of magnesium diglycinate vs magnesium oxide in patients with ileal resection. Journal of Parenteral and Enteral Nutrition, 18(5), 430-435. https://doi.org/10.1177/0148607194018005430

Supakatisant, C., & Phupong, V. (2012). Oral magnesium for relief in pregnancy-induced leg cramps: A randomised controlled trial. Maternal & Child Nutrition, 11(2), 139-145. https://doi.org/10.1111/j.1740-8709.2012.00440.x

Wu, L. J., & Zhuo, M. (2009). Targeting the NMDA receptor subunit NR2B for the treatment of neuropathic pain. Neurotherapeutics, 6(4), 693-702. https://doi.org/10.1016/j.nurt.2009.07.008

Yamadera, W., Inagawa, K., Chiba, S., Bannai, M., Takahashi, M., & Nakayama, K. (2007). Glycine ingestion improves subjective sleep quality in human volunteers, correlating with polysomnographic changes. Sleep and Biological Rhythms, 5(2), 126-131. https://doi.org/10.1111/j.1479-8425.2007.00262.x

Zulli, A. (2011). Taurine in cardiovascular disease. Current Opinion in Clinical Nutrition and Metabolic Care, 14(1), 57-60. https://doi.org/10.1097/MCO.0b013e328340d863