Cravings 101: Understanding the Fundamentals of Why We Crave

What to Know

  • Cravings are learned responses involving reward, stress, memory, habit and the environment.
  • A trigger is the cue, a craving is the urge, and withdrawal is the body’s response to reducing or stopping alcohol after physical dependence.
  • Heavy alcohol use can affect dopamine, GABA, glutamate, stress and opioid systems, but cravings are not caused by one simple neurotransmitter imbalance.
  • Cravings can feel relentless, but they often rise, peak and change. The immediate aim is to create time, distance and support before acting.
  • Recognising your common triggers helps you plan for the moments when judgement and impulse control may be under pressure.
  • If you drink heavily, drink daily or have had withdrawal symptoms, seek medical advice before stopping suddenly.

As I drove home on a Friday evening, travelling down my usual route, I noticed a familiar sensation as I passed the bottle shop.

An intense craving.

My mind started creating reasons to pull in:

Come on, you’ve done so well.

You’ve got it this time.

One night will be fine.

For someone trying to stay sober, these thoughts can sound convincing. They can also reopen a pattern that took months of work to interrupt.

Cravings are one of the major roadblocks in recovery from alcohol or other substance use. They can make us vulnerable and may increase the risk of relapse.

The truth is that even when a craving feels relentless, painful and seemingly endless, the intensity can change.

Understanding what is happening gives us more places to intervene.

Cravings Are Old Wiring

A craving is old wiring: a learned connection between a cue, an expected effect and a familiar response. The pathway may have been useful for escaping discomfort in the short term, but it no longer serves the life you are trying to build.

I used to describe it as the body’s old “source code”.

The brain relies heavily on patterns. If alcohol repeatedly follows stress, finishing work, social anxiety, Friday afternoon or a particular route home, those cues can begin to activate the expectation of drinking.

The pattern can look like this:

  1. Cue: a place, time, person, emotion or thought.
  2. Expectation: alcohol will provide relief, reward, confidence or escape.
  3. Craving: attention narrows and the urge becomes stronger.
  4. Response: drinking, buying alcohol or moving closer to the opportunity.
  5. Short-term outcome: the expected state change arrives.
  6. Learning: the brain strengthens the connection between the cue and the response.

This mechanism can trick us into believing alcohol is the only way to get through a situation.

But it is not.

Once we begin to recognise substance cues, or “triggers” as I prefer to call them, we can understand where cravings come from and build a different response.

For many people, cravings become less frequent or less intense with time, new routines and support. They can still return unexpectedly, so the aim is not to prove that you will never have another urge. It is to know what to do when one appears.

Cravings Are Painful, and Your Brain Can Bargain With You

Cravings often narrow attention towards the expected benefit of drinking while pushing previous consequences into the background. That is why the inner argument can sound so reasonable in the moment.

Before you say, “Getting over cravings is not that easy,” I get it.

Cravings can be freakin’ nasty.

There were many times in my own sober journey when I gave in to an intense craving and ended up in a rough patch.

I completed long stints away from alcohol, only for the pattern to return after one apparently harmless thought:

Maybe I have it under control now.

After six months, the mind might say:

You have proven you do not have a drinking problem.

This is a classic priming scenario. The urgency around the original problem fades, the difficult memories feel less immediate, and the idea of drinking again begins to look safer than it is.

That does not mean the brain is deliberately trying to sabotage you. It means reward, memory, stress and habit systems can place enormous weight on the expected short-term effect while the reflective part of the mind is trying to remember the bigger picture.

Craving is clinically important, but it is not a perfect prediction of what happens next. A person can experience a strong urge without drinking, while relapse can also happen with little conscious craving (Tiffany & Wray, 2012).

A craving is an experience. It is not a command.

Cravings on a Biochemical Level

Alcohol cravings involve several interacting brain systems, including reward learning, stress, memory, motivation and executive control. They cannot be reduced to one chemical being simply “high” or “low”.

Heavy or prolonged alcohol use can affect neurological pathways involving dopamine, glutamate, GABA, stress hormones and endogenous opioid systems.

Dopamine is often called a pleasure chemical, but its role is broader. It helps the brain learn which cues deserve attention and motivates us to pursue expected rewards.

If alcohol repeatedly follows a particular cue, the cue itself can become important.

The drive home. The sound of ice in a glass. A difficult meeting. The first quiet moment after the children go to bed.

Glutamate and GABA are also relevant. GABA has inhibitory effects in the nervous system, while glutamate is a major excitatory neurotransmitter. Repeated heavy alcohol exposure can disrupt the balance and adaptation of these systems, which is particularly important in alcohol withdrawal.

The National Institute on Alcohol Abuse and Alcoholism describes addiction as involving changes across reward, stress, memory and executive-control systems. Alcohol-related cues can become powerful because the brain has learned to associate them with alcohol’s effects (NIAAA, 2025).

For a fuller explanation, read Substance Abuse and Neurotransmitters: Understanding the Neurochemistry Behind Addiction and Sobriety.

Our brain chemistry is only part of the picture.

Poor sleep, missed meals, isolation, ongoing stress and a lack of routine can make emotional regulation harder and leave us more exposed when the usual cue appears. These factors do not cause every craving, and fixing them is not a treatment for alcohol dependence. They are part of the foundation that can either support or strain recovery.

Cravings, Triggers and Withdrawal Are Different

A trigger is the cue, a craving is the urge, and withdrawal is the body’s response to reducing or stopping alcohol after physical dependence has developed. They can overlap, but they are not interchangeable.

Withdrawal may include anxiety, sweating, tremor, nausea, agitation, disrupted sleep and an increased heart rate. Severe alcohol withdrawal can involve seizures or delirium and may be life-threatening.

A person in withdrawal may crave alcohol because drinking can temporarily relieve the symptoms.

If you drink heavily, drink daily, have experienced withdrawal before or are unsure about your risk, speak with a GP or alcohol and other drug service before stopping suddenly. The Australian Government advises that some people need medical support to reduce or quit safely (Australian Government Department of Health, Disability and Ageing, 2024).

For more detail, read Alcohol Withdrawal: The Seesaw Effect of Glutamate and GABA.

Substance Cues or “Triggers”

Triggers are situations, thoughts, emotions and environmental cues that can activate a craving or move us closer to drinking. Learning your patterns makes it easier to plan before impulse and judgement come under pressure.

One thing I noticed across longer periods of sobriety, and while writing about recovery, was that certain scenarios repeatedly intensified cravings.

If we can identify those scenarios, we can improve stress adaptation, change the environment and develop different coping responses.

Research on alcohol cue reactivity has found relationships between responses to alcohol-related cues, subjective craving and relapse-related outcomes, although the strength of these relationships varies between people and studies (Witteman et al., 2015).

The triggers I kept seeing fell into four useful categories.

1. Perceived Opportunity Triggers

Perceived opportunity triggers happen when the body senses that alcohol is available or easy to access.

This might be passing a bottle shop or bar, being home alone, having alcohol in the house, receiving a message from someone you used to drink with, or reaching a particular time of day.

Friday evening was a major example for me. The route, the time and the bottle shop formed a familiar sequence.

Early in recovery, there is no prize for testing yourself unnecessarily. Change the route. Remove alcohol from the house. Plan where you will be during the high-risk period. Make the safer response easier to begin.

2. Attention-Allocating Triggers

Attention-allocating triggers occur when alcohol begins taking up more mental space, which can intensify craving and wear down self-control.

You might replay old drinking experiences, focus on what you think you are missing, plan a possible return to drinking or spend hours arguing with the urge.

The aim is not to force every thought away. It is to notice where attention has gone and redirect it deliberately.

A practical activity, conversation, walk, meal, meeting or change of environment can interrupt the mental loop.

3. Stress Triggers

Stress triggers occur when pressure, conflict, fatigue or emotional discomfort activates the old expectation that alcohol will provide relief.

Stress may come from work, family, finances, poor sleep, loneliness, anxiety or the accumulated load of too many demands.

When alcohol has been used as a coping response for a long time, the brain can continue to reach for that option under pressure. Stress-related craving can involve changes in both reward and stress systems, particularly after repeated heavy use (Sinha, 2013).

This is why stress management is not a fluffy extra in recovery. It is part of relapse prevention.

4. Priming Triggers

Priming triggers appear when someone starts believing they can safely control drinking again, often after a longer period of sobriety.

This is the one where the mind loves to bargain:

It has been six months.

I am different now.

I will only drink occasionally.

For some people with a history of alcohol dependence, returning to alcohol can reactivate craving and old patterns quickly.

If this thought appears, bring another person into the decision before acting. Revisit why you stopped, what alcohol was costing you and what happened the last time controlled drinking was attempted.

I explore these four categories in more depth in Identifying Triggers: How to Recognise and Manage the Cues That Lead to Drinking.

What to Do When a Craving Hits

When a craving arrives, focus on the next safe action. Create time, move away from alcohol and contact support before the urge becomes a plan.

Name It

Say: “This is a craving. It is uncomfortable, but I do not have to act on it.”

You can rate the intensity from 0 to 10 and check again after a few minutes. The purpose is to observe change, not to achieve a perfect score.

Delay the Next Action

Do not try to solve the rest of your life while the craving is loud.

Delay the next action. Set a short timer. Shower. Walk. Prepare food. Start a task that occupies your hands. Call someone.

You are creating room for the intensity to shift.

Change the Environment

Leave the bottle shop car park. Take a different route. Go somewhere alcohol-free. Remove access to money or alcohol if that is appropriate and safe.

You do not need to prove your willpower by sitting beside the trigger.

Surf the Urge

Urge surfing treats the craving like a wave.

Notice where you feel it in the body: tightness, restlessness, heat, salivation or a hollow feeling in the stomach.

Observe the sensations as they build and change without feeding them or trying to force them away.

Mindfulness-based relapse prevention has shown promise as one component of treatment for substance use disorders, although it is not a stand-alone solution for everyone (Bowen et al., 2014).

Check the Basics

Ask whether you are hungry, angry or overwhelmed, lonely or tired.

Eat if you have missed a meal. Drink water. Rest if you can. Contact someone if you are isolated.

Basic care does not replace addiction treatment, but it can reduce extra strain in the moment.

Contact Someone

Choose the person before you need them.

A message can be simple:

I am having a strong craving and need to talk for ten minutes.

This might be a friend, sponsor, family member, counsellor, peer worker, GP or alcohol and other drug service.

Record the Pattern

After the craving changes, write down:

  • what happened beforehand
  • where you were
  • what you felt
  • how strong the urge became
  • what you did
  • what helped

This turns a painful moment into information for the next plan.

When Cravings Keep Returning

Frequent, intense or worsening cravings are a reason to strengthen support, not evidence that recovery is impossible.

A GP, addiction medicine specialist or alcohol and other drug clinician can assess:

  • alcohol intake and withdrawal risk
  • mental health
  • sleep and nutrition
  • medications and other substance use
  • social support
  • relapse patterns
  • whether counselling, peer support or medication may help

Medications such as naltrexone and acamprosate may be considered for some people with alcohol dependence as part of a broader treatment plan. They require individual medical assessment and are not suitable for everyone. The Australian Guidelines for the Treatment of Alcohol Problems provide evidence-based guidance on withdrawal, psychosocial care and relapse-prevention treatment.

A return to drinking is serious, but it does not erase everything you learned. Look at what happened, seek support early and strengthen the plan around the point where it broke down.

The Takeaway

There is no hiding it: when we are trying to get sober, cravings suck.

But they are understandable.

Cravings can involve old wiring, learned cues, reward and stress systems, memory, habit, withdrawal and the body’s current state.

If we remind ourselves that the intensity can change, reduce unnecessary exposure to triggers and practise a different response, we can gradually weaken the old pattern.

Start with the next safe action:

Name the craving. Create distance. Change the environment. Contact someone. Let the intensity move.

Then look beyond the immediate moment. Build routines, plan for the four trigger categories and get the level of support that matches the seriousness of the pattern.

If cravings are frequent, intense or connected with withdrawal symptoms, speak with a GP or alcohol and other drug service. In Australia, the National Alcohol and Other Drug Hotline offers free and confidential information, counselling and referrals on 1800 250 015 (Australian Government Department of Health, Disability and Ageing, 2025).

FAQ

How long does an alcohol craving last?

An alcohol craving has no fixed duration. Some urges rise and fall over minutes, while others return in waves or remain in the background for longer. Focus on whether the intensity is changing and what will help you avoid acting on it.

Why do I crave alcohol at the same time every day?

Craving alcohol at the same time each day often reflects a learned routine. The brain may associate that time with reward, transition or relief, such as drinking after work. Changing the environment and planning another activity before the usual time can interrupt the sequence.

Does craving alcohol mean I am addicted?

Craving is one feature considered in alcohol use disorder, but craving alone does not establish a diagnosis. Assessment also considers loss of control, consequences, tolerance, withdrawal, time spent drinking and the role alcohol plays in your life.

Can alcohol cravings happen after months of sobriety?

Alcohol cravings can occur after months of sobriety when a cue, memory, stressor or familiar environment activates an old learned association. A later craving does not erase recovery progress, but it is worth responding to early and reviewing the trigger.

Can medication reduce alcohol cravings?

Medication may reduce cravings or relapse risk for some people with alcohol dependence. Options such as naltrexone or acamprosate require assessment by a doctor because suitability depends on medical history, current alcohol use, other medications and treatment goals.

Is it safe to stop drinking when cravings are strong?

Strong cravings alone do not determine withdrawal risk. If you drink heavily, drink daily, have had withdrawal symptoms or seizures before, or are unsure about your risk, seek medical advice before stopping suddenly because alcohol withdrawal can be dangerous.

References

Australian Government Department of Health, Disability and Ageing. (2024). How can you reduce or quit alcohol? https://www.health.gov.au/topics/alcohol/about-alcohol/how-can-you-reduce-or-quit-alcohol

Australian Government Department of Health, Disability and Ageing. (2025). National Alcohol and Other Drug Hotline. https://www.health.gov.au/contacts/national-alcohol-and-other-drug-hotline

Australian Guidelines for the Treatment of Alcohol Problems. (2026). Guidelines for the treatment of alcohol problems. https://alcoholtreatmentguidelines.com.au/

Bowen, S., Witkiewitz, K., Clifasefi, S. L., et al. (2014). Relative efficacy of mindfulness-based relapse prevention, standard relapse prevention, and treatment as usual for substance use disorders. JAMA Psychiatry, 71(5), 547–556. https://doi.org/10.1001/jamapsychiatry.2013.4546

National Institute on Alcohol Abuse and Alcoholism. (2025). Neuroscience: The brain in addiction and recovery. https://www.niaaa.nih.gov/health-professionals-communities/core-resource-on-alcohol/neuroscience-brain-addiction-and-recovery

Sinha, R. (2013). The clinical neurobiology of drug craving. Current Opinion in Neurobiology, 23(4), 649–654. https://pubmed.ncbi.nlm.nih.gov/23764204/

Tiffany, S. T., & Wray, J. M. (2012). The clinical significance of drug craving. Annals of the New York Academy of Sciences, 1248, 1–17. https://pubmed.ncbi.nlm.nih.gov/22172057/

Witteman, J., Post, H., Tarvainen, M., et al. (2015). Cue reactivity and its relation to craving and relapse in alcohol dependence: A combined laboratory and field study. Psychopharmacology, 232, 3685–3696. https://pubmed.ncbi.nlm.nih.gov/25439993/

Alcohol Withdrawal: The Seesaw Effect of Glutamate and GABA

Article Snapshot (AI Generated)

  • Chronic alcohol use can create what I like to call the seesaw effect of glutamate and GABA.
  • The more heavily and regularly alcohol is used, the harder the brain may have to work to keep things balanced.
  • When alcohol is suddenly reduced or removed, the opposite effect can happen: the nervous system can become overstimulated.
  • This is one reason alcohol withdrawal can involve anxiety, shaking, sweating, insomnia, agitation and, in more serious cases, seizures or delirium.
  • Alcohol withdrawal can be medically serious. If you have been drinking heavily or for a long time, speak with a medical professional before stopping suddenly.

Chronic alcohol use creates what I like to call the seesaw effect of glutamate and GABA, and the more alcohol is used, the larger the swing can become.

When we wake up the next day, depending on the amount of alcohol consumed, we can often see the opposite of alcohol’s sedating effect.

Instead of relaxed, we feel anxious. Instead of sleepy, we feel wired.

Instead of calm, the nervous system feels like it has been turned up too high.

In this article, I’ll help you understand what alcohol withdrawal actually is, and unpack what’s known as the seesaw effect of glutamate and GABA.

Understanding Alcohol Withdrawal Symptoms

Alcohol withdrawal can happen when someone who has been drinking heavily or regularly suddenly stops drinking or significantly cuts down.

This is not the same as a standard hangover. A hangover is unpleasant, but alcohol withdrawal involves a nervous system that has adapted to alcohol and is now trying to function without it.

Common alcohol withdrawal symptoms may include:

  • anxiety
  • sweating
  • shaking or tremors
  • nausea or vomiting
  • poor appetite
  • poor sleep
  • increased heart rate
  • agitation or irritability
  • sensitivity to light, sound or stress
  • difficulty concentrating

More severe alcohol withdrawal can include seizures, hallucinations, severe confusion or delirium tremens. These symptoms require urgent medical care.

Alcohol withdrawal can also present as a potentially serious syndrome that can occur after abrupt cessation or reduction of heavy, sustained alcohol intake, with symptoms ranging from autonomic hyperactivity and tremor through to seizures and delirium in more severe cases (Jesse et al., 2017).

If you or someone else has severe confusion, seizures, hallucinations, chest pain, severe dehydration or looks seriously unwell, seek urgent medical help. In Australia, call 000 in an emergency. For non-emergency alcohol and drug support, the National Alcohol and Other Drug Hotline is 1800 250 015.

GABA and Glutamate

GABA and glutamate are two important neurotransmitters in the brain, and neurotransmitters are chemical messengers that help our nerve cells communicate.

GABA is generally inhibitory. It slows things down. It is associated with calm, relaxation, sleep and reduced nervous system activity.

Glutamate is generally excitatory. It speeds things up. It is involved in alertness, learning, memory and normal brain activation.

We need a tightly regulated balance of both, otherwise we may feel too sedated or hyper stimulated. Our body is always trying to maintain this balance.

This is where the seesaw analogy will come into play, we’ll discuss a bit more on this soon.

When both sides are balanced, the nervous system can respond appropriately. When one side is pushed too far, the body tries to compensate.

How Alcohol Affects GABA and Glutamate

Alcohol is a central nervous system depressant.

This means it can slow brain and nervous system activity. Part of this effect happens through GABA and glutamate.

Alcohol can increase GABA activity, which is one reason it may initially feel calming, sedating or relaxing. At the same time, alcohol can reduce glutamate activity, which further slows things down.

So, in simple terms:

  • GABA goes up
  • glutamate goes down
  • the nervous system becomes more sedated
  • We generally, feel pretty good!

The downside? Our brain doesn’t like being pushed out of balance.

When alcohol is used repeatedly, the brain starts compensating. It may reduce some GABA-related activity and increase glutamate-related activity to try to keep the nervous system functioning.

Chronic alcohol consumption creates neuroadaptive changes, including downregulation of GABA receptors and increased NMDA receptor expression, which is part of glutamate signaling (Haber & Lintzeris, 2021).

This is the body trying to restore that balance, and when we drink chronically, it’s almost a never ending cycle that worsens over time..

The Seesaw Effect

Think of alcohol as pushing down on one side of the seesaw. We first, experience the lovely, pleasant, sedating effects that come with it – Queue GABA.

When alcohol is removed after a good nights sleep, the sedating effect begins to dissipate.

The brains compensatory mechanics begin to kick in, and some level of anxiety, stimulation or uncomfortable…ness arises – Queue Glutamate.

This is why alcohol withdrawal can feel like the opposite of alcohol intoxication. Alcohol makes us feel relaxed, numb, sometimes sleepy. When we wake up the morning after, we start to feel more anxious, shaky, jittery, and wired..

StatPearls describes alcohol withdrawal as involving a relative reduction in GABA signaling alongside excess glutamate activity, contributing to the hyperactive state seen in withdrawal (Newman & Duby, 2024).

This does not mean every person will have severe withdrawal. It depends on the person’s drinking history, health, previous withdrawal episodes, medications, nutrition status and other factors.

The more we chronically indulge in repeated episodes of drinking though, the larger our seesaw swings, and the worse the symptoms become.

Why Alcohol Withdrawal Can Feel So Uncomfortable

When the nervous system is overstimulated, the symptoms can feel physical, mental and emotional, all at the same time.

Some key withdrawal symptoms you may see (or familiar with) include:

  • shaking hands
  • sweating
  • racing heart
  • nausea
  • poor sleep
  • anxiety
  • irritability
  • agitation
  • panic-like feelings
  • strong cravings

Most of these symptoms are our nervous system trying to recalibrate.

For some people, this process is uncomfortable but manageable with appropriate support. For others, it can become dangerous.

In individuals with severe alcohol withdrawal, this glutamate/GABA ratio gap can widen significantly (Brousse et al., 2012). If alcohol has been used heavily, daily, or for a long time, get medical advice before stopping suddenly.

That is why also to not guess your own risk level from just a simple blog post alone (Yes, even this one).

The Kindling Effect

Another important concept is kindling.

Kindling refers to the way repeated withdrawal episodes may make the nervous system more sensitive over time.

In practical terms, this means repeated cycles of heavy drinking, stopping, withdrawing and drinking again can sometimes make future withdrawal episodes more severe.

Repeated withdrawal experiences can progressively intensify withdrawal symptoms, and this includes an increased risk of seizure vulnerability (Becker, 2014).

It’s one reason why the stop-start cycle becomes so difficult. The swing is so great that we swing ourselves back to alcohol for comfort. We also just try do it alone, which when it comes to quitting alcohol, you should always get help.

For more on the wider neurochemistry involved in alcohol use and recovery, see Substance Abuse and Neurotransmitters: Understanding the Neurochemistry Behind Addiction and Sobriety.

Best Approaches for Alcohol Withdrawal

If you are drinking heavily or think you may be physically dependent on alcohol, the safest first step is to make it known to others.

This can be your loved one, friends, or other related family. You then need to get medical support; speak to your GP or related health professional before stopping suddenly.

This caveat is important here, because if you have been drinking for a long time, you can experience seizures and your life can be at risk.

Once you have spoken with a health professional for medical assessment/monitoring (and medication where appropriate) you can then look at habits, foundational nutrition, and ongoing peer support.

I would recommend looking at the articles below as well to support cravings and getting your foundations on track.

If you’re still unsure, please reach out or book a discovery call.

The Takeaway

Alcohol withdrawal is not simply a rough hangover. It can be the nervous system rebounding after adapting to regular or heavy alcohol use.

GABA and glutamate are only part of the picture, but they give us a useful way to understand why withdrawal can feel shaky, anxious, wired and physically intense.

The brain has been trying to keep the seesaw balanced. When alcohol is suddenly removed, the swing back can be uncomfortable and, for some people, dangerous.

If you are planning to reduce or stop heavy alcohol use, get medical guidance first. With the right support, withdrawal can be managed more safely, and the next stage of recovery can begin from a steadier place.

FAQ

Is alcohol withdrawal the same as a hangover?

No. A hangover usually follows a drinking episode and may include headache, nausea, fatigue and dehydration. Alcohol withdrawal occurs when someone who is physically dependent on alcohol reduces or stops drinking. It can involve nervous system overactivity and may become medically serious.

Why does alcohol withdrawal cause anxiety?

Alcohol can temporarily increase calming GABA-related activity and reduce excitatory glutamate-related activity. With repeated heavy use, the brain adapts. When alcohol is removed, the nervous system can rebound toward overactivity, which may feel like anxiety, agitation, poor sleep or internal restlessness.

Can alcohol withdrawal cause seizures?

Yes. Alcohol withdrawal can cause seizures in some people, especially when there is heavy long-term alcohol use, previous withdrawal seizures or other risk factors. This is one reason medical advice is important before stopping suddenly.

What is delirium tremens?

Delirium tremens, also called alcohol withdrawal delirium, is a severe form of alcohol withdrawal. It can involve confusion, agitation, hallucinations, tremor, unstable blood pressure and other serious symptoms. It is a medical emergency.

What should I do if I want to stop drinking?

Speak with a GP, addiction medicine clinician or alcohol and other drug service before stopping suddenly, especially if you drink heavily or daily. In Australia, you can call the National Alcohol and Other Drug Hotline on 1800 250 015. In an emergency, call 000.

References

Becker, H. C. (2014). Neurochemical mechanisms of alcohol withdrawal. Handbook of Clinical Neurology, 125, 133-156. https://pmc.ncbi.nlm.nih.gov/articles/PMC6943828/

Brousse, G., Arnaud, B., Vorspan, F., Richard, D., Dissard, A., Dubois, M., Pic, D., Geneste, J., Xavier, L., Authier, N., Sapin, V., Llorca, P.-M., De Chazeron, I., Minet-Quinard, R., & Schmidt, J. (2012). Alteration of glutamate/GABA balance during acute alcohol withdrawal in emergency department: A prospective analysis. Alcohol and Alcoholism, 47(5), 501-508. https://doi.org/10.1093/alcalc/ags078

Haber, P. S., & Lintzeris, N. (2021). Alcohol withdrawal. In P. S. Haber & B. C. Riordan (Eds.), Guidelines for the Treatment of Alcohol Problems (4th ed.). Specialty of Addiction Medicine, University of Sydney. https://alcoholtreatmentguidelines.com.au/chapter-8-alcohol-withdrawal

Jesse, S., Brathen, G., Ferrara, M., Keindl, M., Ben-Menachem, E., Tanasescu, R., Brodtkorb, E., Hillbom, M., Leone, M. A., & Ludolph, A. C. (2017). Alcohol withdrawal syndrome: Mechanisms, manifestations, and management. Acta Neurologica Scandinavica, 135(1), 4-16. https://pmc.ncbi.nlm.nih.gov/articles/PMC6084325/

Newman, R. K., & Duby, J. J. (2024). Alcohol withdrawal syndrome. StatPearls. https://www.ncbi.nlm.nih.gov/books/NBK441882/

Unpacking Perimenopause: What Is Happening to Your Hormones and Body

Article Snapshot (AI Generated)

  • Perimenopause is the transition before menopause, when ovulation becomes less consistent and hormone patterns become more unpredictable.
  • Oestrogen does not simply decline in a straight line. It can rise and fall from month to month, while progesterone often becomes lower or less reliable because ovulation is less consistent.
  • This shifting oestrogen/progesterone pattern can explain why symptoms feel random: heavier bleeding, breast tenderness or irritability in one cycle, then hot flushes, night sweats, vaginal dryness or poor sleep in another.
  • FSH and LH are brain messenger hormones that try to keep the ovaries responding. Because the ovaries respond less predictably during perimenopause, these markers can also fluctuate.
  • The first clue is often a period change, but symptoms can also include sleep disruption, anxiety, brain fog, fatigue, libido changes, vaginal dryness, joint pain and body composition changes.
  • A single hormone blood test often does not tell the full story. Symptoms, cycle history and broader health markers usually give more useful context.
  • The best support plan looks at the whole system: sleep, stress, nutrition, blood glucose, muscle, bone health, alcohol intake, medications, medical history and whether MHT or other treatments are appropriate.

Perimenopause feels like your body has changed the rules without telling you.

One month your cycle is predictable, the next, your period is early, late, heavier, lighter, or missing altogether.

Your sleep becomes lighter, anxiety is louder, and you notice brain fog, night sweats, low libido and intolerance to stress.

This all being said, Perimenopause is a normal part in the transition into menopause.

In this article, we’ll unpack further what perimenopause is. We’ll discuss the stages from perimenopause into menopause, how it affects quality of life and some tips of what we can do manage it.

What Is Perimenopause?

Perimenopause is the transition leading up to menopause.

Women are considered to have reached menopause once you have gone 12 months without a period, assuming there is no other medical reason for the bleeding change.

Perimenopause is the lead-up to that point, when ovarian function becomes less consistent and hormone patterns become more variable.

A Simple Way to Understand Oestrogen and Progesterone

Two key hormones involved in this transition are oestrogen and progesterone.

Oestrogen is often thought of as the hormone that supports the first half of the menstrual cycle. It helps build the uterine lining, supports vaginal and urinary tissues, influences mood, temperature regulation, skin, bones, blood vessels and brain function.

Progesterone is mainly made after ovulation. It helps balance the second half of the cycle, supports a calmer nervous system, helps regulate bleeding patterns and can influence sleep, mood and fluid retention.

During perimenopause, oestrogen does not simply fall in a straight line. It can rise and fall unpredictably. Progesterone often becomes lower or less consistent because ovulation becomes less reliable. This can create a feeling of hormonal imbalance: some months may feel like high-oestrogen symptoms, such as heavier bleeding, breast tenderness or irritability, while other months may feel more like low-oestrogen symptoms, such as hot flushes, night sweats, vaginal dryness or poorer sleep.

So when people say hormones are “declining,” it is only part of the story. Perimenopause is better understood as a time of fluctuating oestrogen and less reliable progesterone, before the lower-oestrogen pattern of menopause becomes more established.

When Does Perimenopause Affect Women?

Perimenopause usually begins between ages of 45 to 56 years of age and may last around 4 to 8 years (Peacock & Ketvertis, 2023). Some women move through it with minimal disruption. Others have symptoms for years.

A better way to think about the whole picture is in phases:

  • Early perimenopause: cycles may still come regularly, but they start to change. The luteal phase may shorten, PMS may worsen, sleep may become more fragile, and breast tenderness or heavier bleeding may appear.
  • Mid perimenopause: cycles become more obviously variable. Ovulation may be inconsistent, bleeding can become heavier or less predictable, and symptoms such as hot flushes, night sweats, anxiety and brain fog can become more noticeable.
  • Late perimenopause: cycles may be more than 60 days apart. Oestrogen may be lower more often, FSH may trend higher, and classic menopause symptoms may become stronger.
  • Menopause and postmenopause: menopause is reached after 12 months without a period. Postmenopause is the stage after that.

The First Sign = Period Changes

Usually, the most reliable clue we see to determine perimenopause is changes in your menstrual cycle.

Of course, there are many other reasons why your cycle could be irregular, but this is often the first clue, assuming you’re within the age range (or close to it).

Common menstrual cycle signs include periods becoming irregular, shorter or longer than usual, lighter or heavier than usual, or absent for a few months (Jean Hailes for Women’s Health, 2026.).

Some examples include:

  • cycles becoming shorter, sometimes 25 days or less
  • cycles becoming longer or more spread out
  • heavier bleeding than usual
  • lighter bleeding than usual
  • spotting or unpredictable bleeding
  • more painful periods
  • worse PMS, breast tenderness or irritability
  • occasional skipped periods

One common trap is assuming perimenopause only starts when periods become very irregular, but symptoms can start when cycles still look fairly regular.

While again, a lot of things can contribute to cycle irregularity (such as busy life, high stress or poor diet), it can be easily to blame these and overlook perimenopause all together.

The Unpredictable Nature of Hormones in Perimenopause

Perimenopause is not simply a smooth decline in oestrogen.

Oestrogen fluctuates. Progesterone often becomes less consistent as ovulation changes. Follicle-stimulating hormone (FSH) and luteinising hormone (LH) can also shift as the brain tries to keep the ovaries responding. Some cycles are ovulatory. Some are not.

FSH and LH are messenger hormones from the brain. FSH helps stimulate follicles in the ovaries, while LH helps trigger ovulation. During perimenopause, the ovaries may not respond as predictably as they once did, so the brain can send stronger or more variable signals. This is one reason FSH can look higher in one cycle and lower in another, and why a single blood test may not tell the full story (Hechtman, 2025)..

This leads to some months feeling almost normal, then the next month you feel completely derailed.

This is also why perimenopause can feel a little bit random, because you can have:

  • a normal-looking period but poor sleep
  • heavy bleeding and breast pain one month, then a skipped period later
  • anxiety without hot flushes
  • brain fog before obvious cycle disruption
  • night sweats mainly before your period

Common Perimenopause Symptoms

Perimenopause symptoms can be physical, emotional, cognitive, metabolic and sexual.

Common symptoms include:

  • hot flushes
  • night sweats
  • broken sleep or waking at 2-4 am
  • fatigue
  • headaches or migraines
  • breast tenderness
  • heavier or irregular bleeding
  • vaginal dryness
  • urinary urgency or recurrent irritation
  • lower libido
  • mood swings and low mood
  • anxiety or a reduced tolerance to stress
  • irritability or rage
  • brain fog
  • muscle aches or joint pain
  • changes in body composition
  • increased abdominal weight gain

Perimenopause and menopause affect women differently, with symptoms ranging from hot flushes and sleep problems to anxiety, brain fog and changes to mental health and wellbeing (Australian Government Department of Health, Disability and Ageing, 2026).

Progesterone Often Drops Before Oestrogen

One of the most useful things to understand is progesterone.

Progesterone is mainly produced after ovulation. If ovulation becomes less consistent, progesterone production can become less consistent too. You can still have oestrogen activity, but less progesterone to balance the second half of the cycle.

This can contribute to symptoms such as:

  • heavier bleeding
  • breast tenderness
  • worse PMS
  • anxiety
  • irritability
  • sleep disruption
  • night sweats
  • migraines
  • shorter cycles

Perimenopause is a time when cycles may still be present, but luteal phase changes and lower mid-luteal progesterone can contribute to poorer stress tolerance, anxiety, sleep disurbances, breast pain and heavier bleeding (Skilton, 2026).

In plain English: you may still be cycling, but the quality and rhythm of ovulation can change. When ovulation changes, progesterone changes. When progesterone changes, the nervous system often feels it.

Should I Do Hormone Testing?

Yes and no.

For most women in the expected age range, perimenopause is assessed through symptoms, age and cycle changes. Jean Hailes notes that if you are between 45 and 55, it is not usual for a doctor to measure hormone levels to diagnose perimenopause; symptoms and period changes are usually more useful (Jean Hailes for Women’s Health, 2026).

That can be frustrating if you want a clear answer from a blood test. But it makes sense clinically. FSH, oestradiol and progesterone can vary across the cycle and across different months. A single blood test might catch one moment, not the whole transition, a blood test can be almost useless.

Testing may still be useful when:

  • symptoms start early (Much further before age 45)
  • periods stop much earleir too.
  • Strange, unusual or severe symptoms that are ongoing
  • bleeding is heavy, prolonged or unpredictable
  • pregnancy is possible
  • thyroid disease, iron deficiency, B12 deficiency, vitamin D deficiency, diabetes or other conditions need to be ruled out
  • there is a history of chemotherapy, radiotherapy, ovarian surgery or hysterectomy

A better approach for hormone testing is actually through dried urine, saliva and serum testing, often known as a DUTCH test.

This usually involves multiple collection across a 24-hour period to better understand sex and adrenal hormonal patterns, which can often better help the clinical picture (Hechtman, 2025).

Heavy Bleeding and Perimenopause

Some bleeding changes are common in perimenopause. That does not mean all bleeding changes are harmless.

Always speak to your GP if you experience the following over a consistent basis:

  • very heavy bleeding
  • bleeding that lasts longer than usual
  • bleeding between periods
  • bleeding after sex
  • bleeding after menopause
  • pelvic pain
  • unexplained weight loss
  • dizziness, faintness or symptoms of iron deficiency

Healthdirect recommends medical review if perimenopausal symptoms are bothering you or affecting daily life, and specifically flags heavy bleeding, bleeding between periods and bleeding after sex as reasons to see a doctor (Healthdirect, n.d.).

Heavy bleeding can also contribute to low iron, which can worsen fatigue, breathlessness, restless legs, poor exercise tolerance, hair shedding and low mood. If bleeding has changed significantly, iron studies are often worth discussing.

The Stress Picture

Perimenopause and the ovaries have a substantial link to the brain and nervous system.

The hypothalamic-pituitary-ovarian (HPO) axis is the communication loop between the brain and ovaries. The hypothalamic-pituitary-adrenal (HPA) axis is the communication loop between the brain and adrenal stress response. These systems are connected. Sleep loss, under-eating, overtraining, psychological stress, alcohol, blood sugar swings and chronic illness can cause dysregulation across both these systems.

This point is critical, because often perimenopause symptoms are worsened because when our HPO/HPA Axis is burdened by these factors.

Symptoms such as feeling tired but wired, extreme insomnia or very poor stress resilience become amplified.

Sleep, stress management, circadian rhythm, nutrition and movement are core foundations for supporting the HPA/HPO axis, which in return supports an easy transition into menopause (Skilton, 2026).

The takeaway: If your sleep is broken, your blood glucose is swinging, your caffeine is high, your alcohol intake is creeping up and your training is either too intense or non-existent, perimenopause may feel much worse than it needs to.

Food and Perimenopause

There is no single perimenopause diet, yep, I said it.

There is more key factors to consider when going through perimenopause to implement into (or out of) your current diet.

Some core examples include:

  • enough protein at each meal (This will support your mood, hormones and nervous system in general)
  • fibre-rich carbohydrates such as vegetables, legumes, oats, wholegrains, fruit and root vegetables where tolerated (Think gut health, which also supports mood!)
  • healthy fats from olive oil, avocado, nuts, seeds, eggs and oily fish
  • eat regular meals and do not skip them, this will worsen your anxiety and cravings
  • regular meals if skipping food worsens anxiety, cravings or night waking
  • Eat enough food in general, especially if you are highly active or training intensively
  • reduced alcohol if sleep, hot flushes, mood or weight gain are concerns
  • reduce ultra-processed foods, this increases inflammation and worsens symptoms

If you want it in basic terms, the best approach is eating adequate protein, quality carbohydrates and fat, and limit aggressive dieting.

Protein supports blood glucose stability, thyroid health, lean mass and the amino acid supply needed for hormone-related processes. Carbohydrates can matter too, because low energy availability and low glucose availability may affect LH signalling and ovulation in susceptible women (Skilton, 2026).

This is pretty important. Many midlife women I see respond to body changes by eating less, fasting a lot and pushing 110%. You might move the needle a little bit, but usually, this worsens sleep, stress hormones, cravings and cycle disruption.

For related reading:

Movement and Perimenopause

Perimenopause is a key time to protect muscle and bone, and we can do this through movement.

I know we’re not all on the same level when it comes to exercise motivation, but perimenopause is a pinnacle time where changes happen (and movement becomes important).

Oestrogen changes can affect bone turnover, connective tissue, joint comfort, insulin sensitivity and where body fat is stored. RACGP notes that menopause is associated with reduced bone mineral density and changes in cardiovascular risk factors such as increased central adiposity (Magraith & Jang, 2026).

Research also suggests late perimenopause may be a vulnerable period for skeletal muscle loss. Park et al. found appendicular lean mass was lower in late compared with early perimenopausal women, with FSH proposed as one possible contributor (Park et al., 2020).

Some simple tips to think of movement in your daily routine:

  • Move often and daily, on the hour every hour is good if you have an office job
  • Walk after your lunch break to support healthy blood sugar regulation
  • If possible, incorporate light weights or bodyweight training 20-30 minutes, 2-3 times a week.
  • If difficult, find one single addition you can sustain and repeat daily. This could be just 10-15 minutes of walking, anything extra counts.
  • support vitamin D and calcium status where needed
  • address sleep and alcohol intake
  • avoid long periods of under-eating
  • treat pain or injury properly so you can keep moving

Of course, when it comes to bone, we also need to consider our vitamin D and calcium status, and avoid long periods of under-eating.

For related reading, see Creatine: What It Is, How It Is Used and Why Brain Health Research Is Getting Interesting, Magnesium: Why This Mineral Matters for Energy, Stress, Sleep and the Nervous System and Zinc: Why It Matters for Immunity, Recovery and Overall Health.

Sleep and Perimenopause

Poor sleep can make almost every perimenopause symptom feel worse.

Night sweats can wake you. Anxiety can wake you. Alcohol can fragment sleep. Low blood glucose overnight can wake some people. Stress can make the nervous system more alert when it should be downshifting.

This often flows into the next day, making everything louder, cravings worsen, and mood less steady.

You tend to then drink more coffee, become susceptible to poor food choices, and the list goes on.

So, it’s always good to have a sleep plan to mitigate some of the effects, and support your nervous system as best you can.

Of course, other tailored treatment plans, supplements and herbal medicine can help here too (book a free discovery call to learn how).

Some realistic tactics to support your nervous system (and sleep) might include:

  • 10-15 minutes morning sunlight to better regular your circadian rhythm
  • Setting a consistent wake and bed time (while limiting phone use)
  • caffeine before midday
  • enough food during the day (esp. protein!)
  • alcohol reduction, especially close to bedtime
  • a cooler bedroom
  • A sleep wind down ritual (maybe it’s a tea and reading before bed).

For deeper sleep context, see Sleep Hygiene: What It Is and How to Build Better Sleep Habits and Morning Sun and Circadian Rhythm: Why Light Timing Matters for Sleep, Energy and Mood.

Where Does MHT Fit?

Menopause hormone therapy, or MHT, can be very helpful for some women.

It is not the only option, and it is not automatically suitable for everyone. But it should not be dismissed out of fear either.

RACGP notes that MHT can be offered to symptomatic women where there are appropriate clinical indications and no contraindications, after an individualised discussion about risks and benefits (Magraith & Jang, 2026). It may help symptoms such as hot flushes, night sweats, sleep disturbance and genitourinary symptoms, and it can also have a role in bone health for some women.

The decision depends on your symptoms, age, menstrual stage, uterus status, bleeding pattern, migraine history, cardiovascular risk, clotting history, breast cancer risk, liver health, medications and personal preferences.

This is something you can discuss with your GP, but not something helped with naturopathically. Of course, if you are going through MHT, we can support alongside.

A Practical Start to Perimenopause

If you suspect perimenopause, start with a simple three-month snapshot.

Track the following if you can:

  • cycle length, bleeding volume and duration
  • night sweats or hot flushes (frequency per week, severity)
  • sleep quality (frequency, wake times, time to fall asleep)
  • mood and anxiety
  • migraines or headaches
  • breast tenderness
  • libido and vaginal symptoms (dryness, sex drive)
  • joint pain or muscle aches
  • training tolerance
  • Stress load and tolerance

Useful blood tests to discuss with your GP may include iron studies, thyroid function, fasting glucose or HbA1c, lipids, vitamin D, B12 and other markers depending on your history. Hormones may be tested in some cases, but they are not always the first or most useful step.

This data is so useful for you to bring to a naturopathic consultation too. While we would generally ask questions like this during a consultation, this level of detail gives us much better insight as to what is going on, and allows us to support you better.

Also Consider Some Foundations

  • Eat more regularity and ensure your meals are protein rich
  • lift weights two to three times per week, or just increase movement in general
  • Incorporate an evening sleep ritual (limit screen, drink a tea and read a book)
  • Be kind to yourself and know that this process is a normal transition

The Takeaway

Perimenopause is a hormonal transition, but it is also a whole-body transition.

It can affect cycles, sleep, mood, cognition, libido, bleeding, muscle, bone, blood glucose, stress tolerance and body composition. The pattern can be messy because hormones fluctuate rather than decline in a straight line.

The best approach is not panic, guesswork or random supplements. It is pattern recognition, sensible testing, strong lifestyle foundations and individualised care.

If you want help making sense of your symptoms, nutrition, sleep, blood glucose, stress load or supplement options, Stephen can help you build a practical plan that fits your body, health history and stage of life.

FAQ

What are the first signs of perimenopause?

The first sign is often a change in your menstrual cycle. Periods may become shorter, longer, heavier, lighter, more painful or less predictable. Some women also notice sleep problems, anxiety, night sweats, breast tenderness, migraines or brain fog before their cycle changes dramatically.

Can perimenopause start before 45?

Yes, it can. However, if your periods become irregular or stop before 45, it is worth speaking with your GP. Early menopause and premature ovarian insufficiency need proper assessment.

Why do I feel anxious during perimenopause?

Hormone fluctuations can affect the brain and nervous system. Lower or less consistent progesterone, sleep disruption, stress load, blood glucose swings and life pressure can all contribute. Anxiety in perimenopause is common, but it should still be supported properly.

Do I need a blood test to diagnose perimenopause?

Not always. In the usual age range, perimenopause is generally assessed through symptoms, cycle changes and medical history. Blood tests can still be useful to rule out other causes of symptoms or to assess broader health markers. The better test is a DUTCH test.

Is heavy bleeding normal in perimenopause?

Heavier bleeding can happen in perimenopause, but it should not be ignored. See your GP if bleeding is very heavy, prolonged, occurs between periods, happens after sex or occurs after menopause.

Can nutrition help perimenopause symptoms?

Nutrition can help support energy, mood, blood glucose, muscle, sleep and long-term health. It may not remove every symptom, but adequate protein, fibre-rich carbohydrates, healthy fats and enough total food can make the transition easier for many women.

Should I take supplements for perimenopause?

It depends on your symptoms, health history, medications and goals. Magnesium, creatine, zinc, herbs and other supports may be useful in selected cases, but they should be chosen carefully rather than taken randomly. Book a naturopathic health consultation first and ensure your supplements or herbals are tailored to you.

Is MHT safe?

MHT can be appropriate and helpful for many symptomatic women, but suitability depends on individual risk factors and medical history. Speak with your GP or a menopause-aware doctor to discuss benefits, risks and options.

References

Australian Government Department of Health, Disability and Ageing. (2026). Delivering the first national menopause campaign for Australian women. https://www.health.gov.au/ministers/the-hon-mark-butler-mp/media/delivering-the-first-national-menopause-campaign-for-australian-women

Forbes, S. C., Candow, D. G., Krentz, J. R., Roberts, M. D., & Young, K. C. (2021). Creatine supplementation in women’s health: A lifespan perspective. Nutrients, 13(3), 877. https://pmc.ncbi.nlm.nih.gov/articles/PMC7998865/

Healthdirect. (n.d.). Perimenopause. https://www.healthdirect.gov.au/perimenopause

Hechtman, L. (2025). Supporting patients through perimenopause: EndoMAP Code 1501. Seminar handout. NutriPATH.

Menopause: Symptoms, Causes And Management | Jean Hailes. (2026). In Jean Hailes. https://www.jeanhailes.org.au/health-topics/menopause/

Magraith, K., & Jang, C. (2026). Demystifying menopausal hormone therapy prescribing. Australian Journal of General Practice, 55(4). https://www1.racgp.org.au/ajgp/2026/april/demystifying-menopausal-hormone-therapy-prescribin

Park, Y. M., Jankowski, C. M., Ozemek, C., Hildreth, K. L., Kohrt, W. M., & Moreau, K. L. (2020). Appendicular lean mass is lower in late compared with early perimenopausal women: Potential role of FSH. Journal of Applied Physiology, 128(5), 1373-1380. https://pubmed.ncbi.nlm.nih.gov/32298212/

Peacock, K., & Ketvertis, K. M. (2023). Menopause. In National Library of Medicine. StatPearls Publishing. https://www.ncbi.nlm.nih.gov/books/NBK507826/

Prior, J. C. (2006). Perimenopause lost: Reframing the end of menstruation. Journal of Reproductive and Infant Psychology, 24(4), 323-335. https://doi.org/10.1080/02646830600974071

Skilton, A. (2026). The menopausal transition: A four-phase framework for HPA/HPO axis support. Clinical webinar handout. Designs for Health.

Type 2 Diabetes Explained: What Insulin Resistance Means and What to Do Next

Type 2 Diabetes can sound like a simple blood sugar problem, but there is a bit more going on under the surface.

At its core, Type 2 Diabetes is usually driven by insulin resistance. This means the body is still making insulin, especially earlier on, but the cells are not responding to it as well as they should. Over time, the pancreas may struggle to keep up, blood glucose can stay higher than normal, and the risk of complications can increase.

That sounds heavy, and for some people it is. But it is also worth saying this clearly: Type 2 Diabetes is not a personal failure, and it is not only about willpower. Genetics, age, family history, ethnicity, body composition, sleep, stress, movement, medications, social factors and food environment can all play a role.

In this article, we’ll unpack more of what Type 2 Diabetes is, the difference between Type 1 and Type 2, and what you can do to reverse (or avoid) getting it.

What Is Type 2 Diabetes?

Type 2 Diabetes is a chronic metabolic condition where blood glucose remains higher than it should be. Diabetes Australia describes it as a condition where the body becomes resistant to insulin’s usual effects and gradually loses the capacity to produce enough insulin from the pancreas (Diabetes Australia, n.d.).

When we consume carbohydrates, our body breaks these molecules down into it’s simplest, manageable form known as glucose. This can be also understood as sugar.

Our pancreas then releases insulin, a hormone that knocks on the door of our cells and signals them to absorb this glucose for energy use or storage (found in our muscle or liver).

If we continue to consume carbohydates, and our cells are at full capacity, our cells practically ignore the door knock of insulin. This leads to the transportation of glucose to our liver, where it is converted into fatty acids and stored in our fat. Fortunately, insulin has an almost unlimited storage capacity for this!

As you can imagine over time after excessive stuffing of carbohydrates, our cells begin to ignore insulin further and our fat stores increase. This means, we also have elevated blood sugar for longer, and this is what leads to insulin resistance (or Type 2 Diabetes).

Type 1 and Type 2 Diabetes Are Different

Type 1 and Type 2 Diabetes are often spoken about together, but they are not the same condition.

Type 1 Diabetes is an autoimmune condition where the immune system attacks insulin-producing cells in the pancreas. People with type 1 diabetes need insulin.

Type 2 Diabetes usually develops through a combination of insulin resistance and reduced insulin production over time. Some people with type 2 diabetes may eventually need insulin, but many begin with lifestyle changes, oral medication, injectable medications or a combination of supports depending on their situation.

This matters because, if you do have Type 2 Diabetes, or insulin resistance, it is reversible. Through some simple dietary and lifestyle changes, you can get back on track!

Common Symptoms of Type 2 Diabetes

One frustrating thing about type 2 diabetes is that it can develop quietly, I often see people with some “common” symptoms that are actually Type 2 Diabetes red flags!

Some of these symptoms include increased thirst or urinary frequency, unexplained weight changes or even feeling unusually tired or flat.

We can also experience blurred vision, frequency infections, and even slow-healing of cuts or wounds.

The RACGP lists symptoms such as lethargy, frequent urination, increased thirst, infections, blurred vision, altered sensation, poor wound healing and weight loss as clinical symptoms that may suggest diabetes (RACGP, 2024).

Of course, many of these symptoms are common (such as lethargy) and can overlap as symptoms of iron deficiency. Regardless, if you are experiencing any of these symptoms, please consult with your GP and/or book a discovery call and I can see how I can help.

These symptoms can overlap with many other health issues, so they are not enough to diagnose diabetes on their own. But they are enough to justify a proper conversation with your GP.

Risk Factors of Type 2 Diabetes

The Australian Institute of Health and Welfare groups risk factors into behavioural, biomedical and fixed or non-modifiable factors. Behavioural risk factors may include dietary patterns, physical inactivity and smoking.

Biomedical risk factors may include impaired fasting glucose, impaired glucose tolerance, high blood pressure, dyslipidaemia, overweight, obesity and increased waist circumference. Non-modifiable factors include ageing, sex recorded at birth, family history and ethnic background (AIHW, 2026).

To simplify it, risk of Type 2 Diabetes is influenced primarily by:

  • family history of type 2 diabetes
  • increasing age
  • a history of gestational diabetes
  • polycystic ovary syndrome or signs of insulin resistance
  • higher waist circumference or body weight
  • high blood pressure
  • abnormal cholesterol or triglycerides
  • low physical activity
  • smoking
  • certain medications
  • some ethnic backgrounds
  • previous cardiovascular disease
  • disrupted sleep, chronic stress and broader lifestyle pressures

This does not mean everyone with these risk factors will develop Type 2 Diabetes. Your chances though, can substantially increase if you tick most of these boxes.

Diagnosing Type 2 Diabetes

On top of symptoms, Type 2 Diabetes is further diagnosed through blood tests and often a skin prick.

The tests you’ll commonly see, and the optimal ranges for are below:

  • Fasting blood glucose: a blood glucose test done while fasted to gather how well your body is clearing glucose. An optimal range is 4.44 – 5.55 mmol/L.
  • HbA1c: a marker that reflects average blood glucose over roughly the previous two to three months. An optimal range is 0.041 – 0.057 (4.1% to 5.7%)
  • Oral glucose tolerance test: a test that measures the response to a glucose drink over time, often used when results need more clarification.

Of course, there are more tests that may be done, including the measurement of inflammatory markers, lipid profile or liver function tests.

The most important point is, if you are worried, get tested. You may even wish to go out and buy a Continuous Blood Glucose Monitor, these devices can help you monitor your blood sugar in real time and only cost about $15 to trial.

The important point is simple: if you are worried, get tested. You do not need to wait until symptoms are obvious.

Why Insulin Resistance Matters

Let’s unpack a little more of why Insulin Resistance is a part of the bigger picture, because Insulin Resistance is often diagnosed before Type 2 Diabetes is.

When cells become less responsive to insulin, the body may need to produce more insulin to manage the same amount of glucose. This can happen in muscle, liver and fat tissue. Over time, insulin resistance is often linked with a broader metabolic picture that may include higher blood glucose, blood pressure changes, altered cholesterol, inflammation and increased cardiovascular risk.

This is often why other blood markers are tested (like lipid profile and inflammation), because Type 2 Diabetes care much often becomes less about glucose. The worse it gets, the management of cardiovascular function, kidney function, liver, and long-term health becomes more crucial.

This is not what we want to progress to, and because it is a reversible condition, it’s something I can help with.

Even starting with d daily walk after meals, improving protein intake, reducing ultra-processed foods, building muscle through resistance training and getting better sleep. These sound just like flashy “wellness tips” but they are actually ways to help your body utilise glucose more effectively.

For related nutrition context, you may also find these useful:

Food and Type 2 Diabetes

People often hear “blood sugar” and immediately think they need to cut out every carbohydrate under the sun.

This is a little too reductionist, and provides no real sustainable steps forward.

Carbohydrates do influence blood glucose, but the full meal matters. Fibre, protein, fat, food processing, portion size, timing, sleep, stress, movement and medication can all influence the response.

A practical food pattern for blood glucose support often focuses on:

  • enough protein at meals.
  • Consume fibre rich nutrient dense food over processed food. This means plenty of legumes, vegetables, oats, and intact grains (where tolerated).
  • Always consume plenty of colourful vegetables daily.
  • Do not overconsume fruit (yes, you can consume too much if you’re at risk of diabetes).
  • healthy fats from foods such as olive oil, avocado, nuts, seeds and oily fish
  • fewer sugary drinks and highly processed snack foods
  • Not skipping meals, have breakfast, and ensure your meals are well timed through-out the day.
  • Limit alcohol intake.

For some people, a lower-carbohydrate approach may be useful. For others, a Mediterranean-style pattern, higher-protein pattern, calorie-controlled approach or culturally tailored eating plan may be more realistic.

You find the diet plan, pattern or style that suits you, not what the internet tells you to do.

From a clinic standpoint, matching is highly individualised. This means, all treatment plans prescribed where clinically appropriate, sustainable and matched to you.

Movement Is Helpful

Your muscle is one giant glucose-consuming machine, so the more you use it, the more you help it utilise glucose.

When you move, especially when you use your muscles regularly, glucose has somewhere productive to go. This is one reason walking, strength training and breaking up long periods of sitting can be useful for blood glucose support.

RACGP guidance for adults with Type 2 Diabetes includes aiming for 150 minutes of aerobic activity each week, plus two to three resistance exercise sessions, where appropriate and safe for the individual (RACGP, 2024).

I honestly think this recommendation, while achievable, is not realistic for most people starting out and looking to reverse their condition.

My recommendations for most clients and starting points include:

  • 10-15 minute walk after one meal each day; lunch is usually a good one here.
  • Two simple 20-30 minute training sessions a week (this could even just be bodyweight at home).
  • Moving or standing up every hour on the hour, especially if you sit at work.
  • Gradually increasing your steps (if you track it) over the day (incremental is key).

It’s best to note, if you have diabetes complications, cardiovascular disease, dizziness, neuropathy, foot problems or have been inactive for a long time, get professional guidance first.

This means seeing your GP, and finding a professional in physical movement (such as a personal trainer or exercise physiologist).

What is Type 2 Diabetes “Remission”

This generally means blood glucose markers have returned below the diabetes range for a sustained period without glucose-lowering medication, though definitions can vary.

Remission is always possible for people with Type 2 Diabetes, particularly when some foundational diet and movement strategies are implemented and sustained over a period of time.

Ongoing monitoring does still matter though to ensure full remission, and this will depend on the length of time, age and other various factors present in the individual.

Medication Is Not a Failure

As a natural health specialist, it can be shocking for me to say this but, there is always a time and place for medication. If you are having astronomically high blood glucose readings, your health is at risk and you need medication in the short-term to save your life.

The Australian Diabetes Society’s 2026 glycaemic management algorithm notes that treatment for Type 2 Diabetes should be individualised, including appropriate blood glucose and HbA1c targets that consider the person’s situation and preferences (Australian Diabetes Society, 2026).

What this essentially means is, some people really do need medication early, and some need tapered support over time, it’s all individualised.

Most clients who come to me are already on medication (and that is completely okay). I work with your GP, your medication, and we put together a broader plan to work on your food, movement, and any other targeted areas of support.

When to Get Checked

Speak with your GP or healthcare provider if you have symptoms, a family history of diabetes, a history of gestational diabetes, high blood pressure, abnormal cholesterol, PCOS, unexplained fatigue, frequent infections, increased thirst or urination, or concerns about your blood glucose.

You should seek medical advice promptly if symptoms are significant, worsening or accompanied by unexplained weight loss, vomiting, confusion, severe dehydration, chest pain or shortness of breath.

If you already have Type 2 Diabetes, do not change prescribed medication, insulin, alcohol intake around medication, or carbohydrate intake dramatically without clinical guidance.

FAQ

What is type 2 diabetes in simple terms?

Type 2 Diabetes is a condition where blood glucose stays too high because the body does not respond to insulin properly and may eventually struggle to make enough insulin. This is often linked with insulin resistance.

What are the first signs of Type 2 Diabetes?

Possible signs include tiredness, increased thirst, passing urine more often, blurred vision, slow wound healing, frequent infections and tingling or numbness in the hands or feet. Some people have no obvious symptoms and are diagnosed through blood tests.

Can Type 2 Diabetes be prevented?

Some cases may be delayed or prevented, especially when modifiable risk factors are addressed early. Helpful areas can include nutrition, physical activity, weight management, smoking cessation, sleep and regular screening for people at higher risk.

Can Type 2 Diabetes go into remission?

Some people can achieve remission, particularly with early and structured changes that improve blood glucose and reduce metabolic strain. Remission is not the same as a permanent cure, and ongoing monitoring is still important.

Do I need to cut out all carbohydrates if I have Type 2 Diabetes?

Not necessarily. Carbohydrate quality, portion size, fibre, protein, meal timing, activity and medication all matter. Some people do well with lower-carbohydrate approaches, while others need a different plan. It is best to personalise this with a qualified healthcare professional.

Is Type 2 Diabetes only caused by being overweight?

No. Body weight can be one risk factor, but type 2 diabetes is influenced by genetics, age, ethnicity, family history, body fat distribution, physical activity, blood pressure, cholesterol, gestational diabetes history, medications and other factors.

When should I get tested for Type 2 Diabetes?

If you have symptoms or risk factors, speak with your GP. In Australia, routine risk assessment is recommended from age 40 for people without specific risk factors, and earlier or more regular testing may be recommended for higher-risk groups.

References

Australian Diabetes Society. (2026). Australian Type 2 Diabetes Glycaemic Management Algorithm (May 2026). https://www.diabetessociety.com.au/guideline/australian-t2d-glycaemic-management-algorithm-june-2024/

Australian Institute of Health and Welfare. (2026). Diabetes: Australian facts – Risk factors for diabetes. https://www.aihw.gov.au/reports/diabetes/diabetes/contents/diabetes-risk-factors

Diabetes Australia. (n.d.). Type 2 diabetes. https://www.diabetesaustralia.com.au/about-diabetes/type-2-diabetes/

Diabetes UK. (n.d.). Type 2 diabetes. https://www.diabetes.org.uk/about-diabetes/type-2-diabetes

Royal Australian College of General Practitioners. (2024). Defining and diagnosing type 2 diabetes. https://www.racgp.org.au/clinical-resources/clinical-guidelines/key-racgp-guidelines/view-all-racgp-guidelines/management-of-type-2-diabetes/defining-and-diagnosing-type-2-diabetes

Royal Australian College of General Practitioners. (2024). Type 2 diabetes: Goals for optimum management. https://www.racgp.org.au/clinical-resources/clinical-guidelines/key-racgp-guidelines/view-all-racgp-guidelines/management-of-type-2-diabetes/type-2-diabetes-goals-for-optimum-management

Royal Australian College of General Practitioners. (2024). Lifestyle interventions for the management of type 2 diabetes. https://www.racgp.org.au/clinical-resources/clinical-guidelines/key-racgp-guidelines/view-all-racgp-guidelines/management-of-type-2-diabetes/lifestyle-interventions-for-the-management-of-type

Digestive Health and Addiction: Why Gut Health Matters After Quitting Alcohol

Excessive alcohol intake is usually discussed in relation to the brain, liver, cravings, sleep and behaviour. But alcohol can also significantly impact our digestive system.

Digestive health influences how we break down food, absorb nutrients, eliminate waste and communicate with the nervous system. When digestion is poor, people may notice bloating, reflux, diarrhoea, constipation, nausea, low appetite or unpredictable hunger cues. Those symptoms can make early sobriety feel harder than it needs to.

This does not mean gut health is a long-term fix for addiction; sobriety is bigger than digestion alone.

In this article, we’ll unpack why digestive health matters after quitting alcohol, the significance of the Gut-Brain Connection, and tips to support our gut health when quitting alcohol.

Why Digestive Health Matters After Quitting Alcohol

The digestive system is responsible for breaking down food, absorbing nutrients and removing waste from the body.

When someone has been drinking heavily or frequently, nutrition often takes a hit. Meals may become inconsistent. Appetite can change. Alcohol may displace more nutrient-dense foods. Some people also experience digestive symptoms that make eating well more difficult.

This is why digestive health and nutrition become important when we look at addiction and sobriety.

If our gut health is irritated, inflamed or simply not functioning as it should, it can make it harder to replenish nutrients and support long-term sobriety.

For more on nutrient depletion and alcohol, read Alcohol and Nutrition: Does Chronic Drinking Cause Nutrient Deficiency?. If you are rebuilding meals, the Protein Sources Guide is a practical next step.

Understanding the Gut-Brain Connection

The gut and brain are in constant communication. This is often called the gut-brain axis.

That communication happens through the nervous system, immune signalling, hormones, microbial metabolites and inflammatory pathways. In plain English, the gut does not just digest food. It also sends and receives signals that can influence how the body feels.

The Two-Way Communication Pathway

Stress can affect digestion. Digestion can affect comfort, mood and energy. Anyone who has lost their appetite during anxiety or felt gut symptoms during stress has experienced this connection in a very practical way.

When quitting alcohol, this matters because the body and brain are both recalibrating. Sleep, cravings, blood sugar, appetite, mood and stress tolerance can be all shifting at once.

Research on the microbiome-gut-brain axis in substance-related disorders is still developing, but it is a meaningful area of interest. Fu et al. (2021) describe the gut-brain axis as a potential therapeutic research target in substance-related disorders, while also making it clear this is not a simple one-cause, one-cure relationship.

The Role of Gut Microbiota

The gut microbiota is the community of bacteria, fungi and other microorganisms living in the digestive tract. These microbes help with digestion, immune function, gut barrier health and the production of certain metabolites.

Alcohol can disturb this microbial balance, a pattern often called dysbiosis. A meta-analysis by Qamar et al. (2019) reviewed links between alcohol-induced gut dysbiosis and behavioural effects, including pathways connected to inflammation, mood and addiction biology.

The takeaway here is our gut health may influence how we feel in alcohol recovery.

For more on the brain side of addiction recovery, read Substance Abuse and Neurotransmitters.

How Alcohol Can Affect Digestive Health

Alcohol can affect the digestive system in several ways.

It may irritate the stomach lining, contribute to reflux or nausea, and worsen diarrhoea or abdominal discomfort in some people. Over time, heavier alcohol use can also affect the liver, pancreas and the broader gut-liver axis.

Alcohol can also influence the gut barrier. The gut lining is meant to be selectively permeable: it allows useful nutrients through while helping keep unwanted substances out. Chronic alcohol exposure has been linked with gut barrier disruption, increased intestinal permeability and gut-derived inflammation (Bishehsari et al., 2017).

This is one reason the term “leaky gut” appears so often online, you’ve probably seen it if you’ve rabbit-holed down the path of gut health before.

The phrase can be oversimplified at times, but the underlying research around alcohol, intestinal permeability, inflammation and microbiome changes are real. Wang et al. (2020) reviewed how alcohol addiction may interact with gut microbiota and inflammatory pathways.

This does not mean though that every digestive symptom after quitting alcohol is caused by the microbiome. Symptoms can have many causes, and persistent or severe symptoms deserve proper medical assessment.

If you are not sure what’s going on and digestion is a major issue for you, book a FREE discovery call here.

Supporting Digestive Health After Quitting Alcohol

There are many ways to support digestion after alcohol, but the best starting point is usually simple.

Rather than jumping straight to complicated supplement protocols, or comprehensive, often confusing diets, start with the foundations.

Eat a Balanced Diet

Aim for regular meals built around vegetables, fruit, wholegrains, legumes, adequate protein and healthy fats.

Fibre is especially useful because it supports bowel regularity and provides fuel for beneficial gut bacteria. Increase it gradually, especially if you are prone to bloating.

And no, this does not mean consuming copious amounts of Metamucil or Psyllium husk powder. This means a diverse range of fruit and vegetables too.

Protein also matters during recovery because it supports tissue repair, immune function, appetite regulation and neurotransmitter production. The Protein Intake for Exercise article explains this in more detail.

Use Fermented and Prebiotic Foods Carefully

Fermented foods may help some people support gut health. Options include yoghurt with live cultures, kefir, sauerkraut, kimchi, miso, tempeh and kombucha.

Prebiotic foods help feed beneficial gut bacteria. These include oats, legumes, onion, garlic, leek, asparagus, slightly green banana and some wholegrains.

These foods are useful, but they are not magic. Some people with reflux, bloating or histamine-type symptoms may not tolerate fermented foods well. Start small and pay attention to your own response.

Manage Stress

Stress can change appetite, gut motility, stomach comfort and bowel habits. This is why digestive support after alcohol is not only about food.

Helpful basics include eating slowly, chewing properly, walking after meals, reducing rushed eating, using breathing exercises, and getting support for anxiety or emotional stress when needed.

If cravings and triggers are part of the recovery picture, Identifying Triggers may help you think through the behavioural side more clearly.

Prioritise Sleep

Sleep is essential for recovery. Poor sleep can affect appetite, cravings, mood, blood sugar regulation and digestive comfort.

Good sleep does not fix everything, but it gives the nervous system a better foundation. Start with a consistent wake time, morning light, reduced late caffeine and a calmer evening routine. You can read more in Sleep Hygiene and Morning Sun and Circadian Rhythm.

Tips to Support Your Gut Post-Alcohol

Start with a plan that is boring enough to follow:

  • Eat regular meals instead of skipping food and grazing randomly.
  • Include protein at breakfast, lunch and dinner.
  • Increase fibre slowly through vegetables, oats, legumes, berries and wholegrains.
  • Drink enough water across the day.
  • Be mindful of alcohol replacements such as excess sugar, soft drink, energy drinks or too much caffeine.
  • Add fermented foods only if you tolerate them well.
  • Walk after meals when possible.
  • Get medical help if symptoms are severe, persistent or unusual.

It is also important to seek medical advice before suddenly stopping alcohol if you have been drinking heavily or daily. Alcohol withdrawal can be dangerous for some people and may need supervised care.

When to Get Help

Digestive changes can happen after quitting alcohol, but not everything should be brushed off as recovery.

Speak with a doctor or qualified health professional if you notice blood in the stool, black stools, ongoing vomiting, severe abdominal pain, unexplained weight loss, persistent diarrhoea or constipation, yellowing of the skin or eyes, difficulty swallowing, dehydration, or symptoms that do not settle with basic changes.

The Takeaway

Good digestive health can support the bigger recovery picture after quitting alcohol.

Alcohol may affect the stomach lining, gut microbiome, intestinal barrier, inflammation and nutrient absorption. Recovery gives the body a chance to rebuild, but the basics still matter: regular meals, enough protein, gradual fibre intake, hydration, sleep, stress management and proper care when symptoms need attention.

You do not need a perfect gut health protocol. Start with simple, consistent habits and build from there.

If you are rebuilding your health after alcohol and want help making sense of nutrition, recovery habits and sustainable lifestyle change, Stephen can help you take a practical, evidence-informed approach that fits real life.

FAQ

Can quitting alcohol improve digestion?

Of course it can! Many people notice digestion improves after reducing or stopping alcohol, especially if alcohol was contributing to reflux, nausea, diarrhoea, poor appetite or gut irritation. The timeline varies.

How does alcohol affect the gut microbiome?

Alcohol may alter the balance of gut microbes and contribute to dysbiosis, particularly with heavier or chronic intake. It can also affect the gut lining, immune signalling and inflammation.

Are probiotics useful after quitting alcohol?

They may help some people, but they are not a guaranteed fix. Food quality, fibre intake, sleep, stress and overall health habits are usually a better starting point. If in doubt, it’s always best to consult with a Nutritionist, Naturopath or Health Professional (like myself!).

Is gut health linked to cravings?

The gut-brain axis is an active area of addiction research, but I couldn’t definitively say that gut health = impact alcohol cravings alone. Better nutrition and digestion may support mood, energy and stability, which can help the broader alcohol recovery process.

References

Bishehsari, F., Magno, E., Swanson, G., Desai, V., Voigt, R. M., Forsyth, C. B., & Keshavarzian, A. (2017). Alcohol and gut-derived inflammation. Alcohol Research: Current Reviews, 38(2), 163-171. https://pmc.ncbi.nlm.nih.gov/articles/PMC5513683/

Engen, P. A., Green, S. J., Voigt, R. M., Forsyth, C. B., & Keshavarzian, A. (2015). The gastrointestinal microbiome: Alcohol effects on the composition of intestinal microbiota. Alcohol Research: Current Reviews, 37(2), 223-236. https://pmc.ncbi.nlm.nih.gov/articles/PMC4590619/

Fu, X., Chen, T., Cai, J., Liu, B., Zeng, Y., & Zhang, X. (2021). The microbiome-gut-brain axis, a potential therapeutic target for substance-related disorders. Frontiers in Microbiology, 12, 738401. https://doi.org/10.3389/fmicb.2021.738401

Qamar, N., Castano, D., Patt, C., Chu, T., Cottrell, J., & Chang, S. L. (2019). Meta-analysis of alcohol induced gut dysbiosis and the resulting behavioral impact. Behavioural Brain Research, 376, 112196. https://doi.org/10.1016/j.bbr.2019.112196

Wang, S.-C., Chen, Y.-C., Chen, S.-J., & Lee, C.-H. (2020). Alcohol addiction, gut microbiota, and alcoholism treatment. International Journal of Molecular Sciences, 21(17), 6413. https://pmc.ncbi.nlm.nih.gov/articles/PMC7504034/

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